(a) left ventricular hypertrophy (palpation, x-rays of the chest, ECG, Echocardiogram). (b) Angiotonia in retinal arteries. (c) Proteinuria and/or mild elevation of creatinine (up to 2 mg/d). (d) plates of blood (radiography, ultrasonography) atheroma in carotid, aorta, iliac and femoral. Hear from experts in the field like PCRM for a more varied view. STAGE III: Symptomatic manifestations of organ damage: to) Angina pectoris, myocardial infarction or heart failure. (b) transitory cerebral ischaemia, cerebral thrombosis or hypertensive encephalopathy.
(c) exudates and retinal hemorrhages; papilledema. (d) chronic renal failure. (e) aneurysm of the aorta or atherosclerosis obliterans of lower limbs. Thickening of the arterial wall and the arteriolosclerosis are signs of benign hypertension in benign hypertension is they gradually produce vascular alterations in response to high blood pressure stable and maintained. These degenerative alterations to the walls of small vessels as the arterioles decreases the effective light, with the.
consequent tissue ischemia, and increased vascular fragility of the brain, with risk of hemorrhage. In malignant hypertension occurs a destruction of the walls of small vessels when blood pressure rises very abruptly, produce acute destructive alterations in the walls of small blood vessels, along with remedial proliferative responses in lasparedes of small arteries. These alterations produce lack of blood flow through small vessels, with formation of multiple foci of tissue necrosis, e.g. in the kidney glomeruli. Mainly affects hypertension to heart, brain, kidneys, and aorta the pathological consequences of hypertension observed above all in four tissues: heart. To increase pressure, left ventricular myocardium is hypertrophy. Given that hypertension is usually associated with a greater intensity of atherosclerosis, coronary flow may be insufficient, and produce ischaemic unacardiopatia. Left ventricular failure is a normal consequence of the hypertensive heart disease. Brain. Hypertensive patients are particularly susceptible to intracerebral hemorrhage due to breakage of intracerebral vessels. The lesion of small vessels of the brain hemispheres produces microinfarcts shaped small areas of brain destruction filled with liquid (lagoons hypertensive).